Vitamin D deficient rickets and genetic Vitamin D resistant rickets<br/>diagnose by ( ) A: epiphyseal changes in X ray B: increased serum alkaline phosphatase C: hypocalcaemia. Hypophosphatemia D: healing appears with treatment of vitamin D 600,000 IU

The<br/>clinical feature of the sequelae stage of vitamin D deficiency<br/>rickets is () A: Skeleton<br/>deformity B: Abnormal<br/>epiphyseal of long bone C: Serum<br/>calcium and phosphorus decrease D: Alkaline<br/>phosphatase decrease E: Irritability,<br/>hidrosis

The following genes ____________________ for carbohydrate metabolism are housekeeping genes in humans. A: lactate dehydrogenase (LDHA),GDP-L-fucose synthetase (TSTA3), & fructose-bisphosphate aldolase (aldolase A, ALDA) B: enolase phosphatase (ENOPH1) & triose-phosphate isomerase (TPI) C: phosphoglycerate kinase (PGK1) & glycogen synthase kinase-3 (GSK3A/B) D: transaldolase in pentose shunt (TALDO1) & phosphoglycerate mutase (PGM)

The following genes ____________________ for the regulation of cell cycle are housekeeping genes in humans. A: Rho GTPase-activating protein 5 (ARHGAP5) & Rho GDP-dissociation inhibitor 1 (ARHGDIA) B: Rho guanine nucleotide exchange factor 7/11/40 (ARHGEF7/11/40) & Ras-related protein Rab-10 (RAB10) C: centromere protein B (CENPB), centromere protein T (CTBP1), & E3 ubiquitin-protein ligase (CCNB1IP1) D: cyclin-K (CCNK), cyclin-H (CCNH), & protein phosphatase 1 regulatory subunit 7 (PPP1R7)