• 2022-06-11 问题

    A neoplastic overgrowth of white blood cells is called: A: anemia B: leukemia C: fibrosis D: cystitis

    A neoplastic overgrowth of white blood cells is called: A: anemia B: leukemia C: fibrosis D: cystitis

  • 2022-06-06 问题

    Which of the following is NOT the basic pathological changes of chronic viral hepatitis() A: Inflammation B: Cirrhosis C: Fibrosis D: Cell apotosis

    Which of the following is NOT the basic pathological changes of chronic viral hepatitis() A: Inflammation B: Cirrhosis C: Fibrosis D: Cell apotosis

  • 2022-06-06 问题

    Which of the following is NOT the basic pathological changes of viral hepatitis() A: Inflammation: Lymphocytes, plasma cells in portal tract B: Damages, Degeneration and Cell death C: Fibrosis D: Cell apotosis

    Which of the following is NOT the basic pathological changes of viral hepatitis() A: Inflammation: Lymphocytes, plasma cells in portal tract B: Damages, Degeneration and Cell death C: Fibrosis D: Cell apotosis

  • 2022-06-01 问题

    In the following lesions, the liver dullness boundary can be enlarged A: Flatulence of gastrointestinal tract B: Acute severe hepatitis C: Liver abscess D: Advanced cirrhosis E: Right pulmonary fibrosis

    In the following lesions, the liver dullness boundary can be enlarged A: Flatulence of gastrointestinal tract B: Acute severe hepatitis C: Liver abscess D: Advanced cirrhosis E: Right pulmonary fibrosis

  • 2022-07-29 问题

    一个肿性纤维化(cystic fibrosis)病人,其细胞中的囊性纤维化跨膜传导调节因子(cystic fibrosis transmembrane conductance regulator,CFTR)基因发生了一个三核苷酸删除突变,导致CFTR蛋白的第508位苯丙氨酸残基删除,继而使该突变蛋白在细胞内折叠错误。该病人体内细胞识别到该突变蛋白,并通过添加泛素分子对其进行修饰,试问被修饰后的突变蛋白的命运如何 A: 泛素将校正突变的影响,从而使突变蛋白的功能恢复正常 B: 分泌到细胞外 C: 进入贮存装泡 D: 被蛋白酶体降解 E: ​被细胞内的酶修复

    一个肿性纤维化(cystic fibrosis)病人,其细胞中的囊性纤维化跨膜传导调节因子(cystic fibrosis transmembrane conductance regulator,CFTR)基因发生了一个三核苷酸删除突变,导致CFTR蛋白的第508位苯丙氨酸残基删除,继而使该突变蛋白在细胞内折叠错误。该病人体内细胞识别到该突变蛋白,并通过添加泛素分子对其进行修饰,试问被修饰后的突变蛋白的命运如何 A: 泛素将校正突变的影响,从而使突变蛋白的功能恢复正常 B: 分泌到细胞外 C: 进入贮存装泡 D: 被蛋白酶体降解 E: ​被细胞内的酶修复

  • 2022-07-29 问题

    一个囊肿性纤维化(cystic<br/>fibrosis)患者,其细胞中的囊性纤维化跨膜传导调节因子(Cystic<br/>Fibrosis Transmembrane Conductance<br/>Regulator,CFTR)基因发生了一个三核苷酸删除突变,导致CFTR蛋白的第508位苯丙氨酸残基删除,继而使该突变蛋白在细胞内折叠错误。该患者体内细胞识别到该突变蛋白,并通过添加泛素分子对其进行修饰。经修饰后的突变蛋白的命运是() A: 泛素将校正突变的影响,从而使突变蛋白的功能恢复正常 B: 分泌到细胞外 C: 进入贮存囊泡 D: 被蛋白酶体降解 E: 被细胞内的酶修复

    一个囊肿性纤维化(cystic<br/>fibrosis)患者,其细胞中的囊性纤维化跨膜传导调节因子(Cystic<br/>Fibrosis Transmembrane Conductance<br/>Regulator,CFTR)基因发生了一个三核苷酸删除突变,导致CFTR蛋白的第508位苯丙氨酸残基删除,继而使该突变蛋白在细胞内折叠错误。该患者体内细胞识别到该突变蛋白,并通过添加泛素分子对其进行修饰。经修饰后的突变蛋白的命运是() A: 泛素将校正突变的影响,从而使突变蛋白的功能恢复正常 B: 分泌到细胞外 C: 进入贮存囊泡 D: 被蛋白酶体降解 E: 被细胞内的酶修复

  • 1